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Dr John Rogers

Dr John Rogers

Lecturer (retired)

Office Phone: +44 (0) 1223 333865, Fax: +44 (0) 1223 333840

Research areas


Research Interests

Neural injury and regeneration. Genetic manipulation promoting axon regeneration. Responses of the mammalian CNS to injury. Viral vectors for repairing damaged neurones.

I am also Jupiter Section Director in the British Astronomical Association.


Prof  James Fawcett (Cambridge Centre for Brain Repair)

Key Publications

Alves JN, Muir EM, Andrews MR, Ward A, Michelmore N, Dasgupta D, Verhaagen J, Moloney EB, Keynes RJ, Fawcett JW, Rogers JH, (2014), AAV vector-mediated secretion of chondroitinase proves a sensitive tracer for azonal arborisations, J. Neurosci. Methods, 227:107-120

Bartus K, James ND, Didangelos A, Bosch KD, Verhaagen J, Yanez-Munoz RJ, Rogers JH, Schneider BL, Muir EM, Bradbury EJ, (2014), Large-scale chondrotin sulfate proteoglycan digestion with chondroitinase gene therapy leads to reduced pathology and modulates macrophage phenotype following spinal cord contusion injury, J. Neuroscience, 34(14):4822-4836

Kanno H, Pressman Y, Moody A, Berg R, Muir ER, Rogers JH, Ozawa H, Ito E, Pearse DD, Bunge MB, (2014), Combination of engineered Schwann cell grafts to secrete neurotrophin and chondroitinase promotes axonal regeneration and locomotion after spinal cord injury, J. Neuroscience, 34(5):1838-1855

Rogers JH, Rhodes K, Roberts R, Raza M, Muir EM, Fawcett JW, Scarpini CG, and Efstathiou, (2003), A herpesvirus vector can transduce axotomized brain neurons, Exp. Neurol., 183, 548-558

Above: Neurons in tissue culture (green) expressing β-galactosidase (red).

Above: Ganglia in tissue culture in which many neurons are expressing β-galactosidase (blue).

Above: Neurons in adult rat brain, in the parabrachial nucleus, expressing c-Jun (green) as a result of experimental axotomy, and β-galactosidase (red).